Targeted Inhibition of Interferon-γ-dependent Intercellular Adhesion Molecule-1 (ICAM-1) Expression Using Dominant-Negative Stat1
Open Access
- 1 November 1997
- journal article
- Published by Elsevier
- Vol. 272 (45), 28582-28589
- https://doi.org/10.1074/jbc.272.45.28582
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Dominant Negative Stat3 Mutant Inhibits Interleukin-6-induced Jak-STAT Signal TransductionJournal of Biological Chemistry, 1996
- Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral DiseaseCell, 1996
- Targeted Disruption of the Stat1 Gene in Mice Reveals Unexpected Physiologic Specificity in the JAK–STAT Signaling PathwayCell, 1996
- Stat1 Depends on Transcriptional Synergy with Sp1Published by Elsevier ,1995
- The Jak Kinases Differentially Associate with the α and β (Accessory Factor) Chains of the Interferon γ Receptor to Form a Functional Receptor Unit Capable of Activating STAT Transcription FactorsJournal of Biological Chemistry, 1995
- TRANSCRIPTIONAL RESPONSES TO POLYPEPTIDE LIGANDS: The JAK-STAT PathwayAnnual Review of Biochemistry, 1995
- Stat recruitment by tyrosine-phosphorylated cytokine receptors: An ordered reversible affinity-driven processImmunity, 1995
- A STAT protein domain that determines DNA sequence recognition suggests a novel DNA-binding domain.Genes & Development, 1995
- Requirement of Serine Phosphorylation for Formation of STAT-Promoter ComplexesScience, 1995
- Ras-Independent Growth Factor Signaling by Transcription Factor Tyrosine PhosphorylationScience, 1993