The Absence of ABCA1 Decreases Soluble ApoE Levels but Does Not Diminish Amyloid Deposition in Two Murine Models of Alzheimer Disease
Open Access
- 1 December 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (52), 43243-43256
- https://doi.org/10.1074/jbc.m508781200
Abstract
No abstract availableKeywords
This publication has 95 references indexed in Scilit:
- Functions of lipoprotein receptors in neuronsJournal of Lipid Research, 2004
- Amyloidogenic processing of the Alzheimer β-amyloid precursor protein depends on lipid raftsThe Journal of cell biology, 2003
- Apolipoprotein E, Amyloid, and Alzheimer DiseaseMolecular Interventions, 2002
- Apolipoprotein E: Far More Than a Lipid Transport ProteinAnnual Review of Genomics and Human Genetics, 2000
- Apolipoprotein E facilitates neuritic and cerebrovascular plaque formation in an Alzheimer's disease modelAnnals of Neurology, 2000
- Substitutions at codon 22 of Alzheimer's A beta peptide induce conformational changes and diverse apoptotic effects in human cerebral endothelial cellsJournal of Biological Chemistry, 2000
- ALZHEIMER'S DISEASE: Genetic Studies and Transgenic ModelsAnnual Review of Genetics, 1998
- Lack of apolipoprotein E dramatically reduces amyloid β-peptide depositionNature Genetics, 1997
- Characterization of the Binding of Amyloid‐β Peptide to Cell Culture‐Derived Native Apolipoprotein E2, E3, and E4 Isoforms and to Isoforms from Human PlasmaJournal of Neurochemistry, 1997
- Gene Dose of Apolipoprotein E Type 4 Allele and the Risk of Alzheimer's Disease in Late Onset FamiliesScience, 1993