RuvABC‐dependent double‐strand breaks in dnaBts mutants require RecA
Open Access
- 1 November 2000
- journal article
- research article
- Published by Wiley in Molecular Microbiology
- Vol. 38 (3), 565-574
- https://doi.org/10.1046/j.1365-2958.2000.02152.x
Abstract
Replication fork arrest can cause DNA double‐strand breaks (DSBs). These DSBs are caused by the action of the Holliday junction resolvase RuvABC, indicating that they are made by resolution of Hollid...Keywords
This publication has 45 references indexed in Scilit:
- DNA supercoiling — a global transcriptional regulator for enterobacterial growth?Nature Reviews Microbiology, 2005
- Modulation of RNA Polymerase by (p)ppGpp Reveals a RecG-Dependent Mechanism for Replication Fork ProgressionCell, 2000
- DNA recombination: the replication connectionTrends in Biochemical Sciences, 1999
- Duplex opening by primosome protein PriA for replisome assembly on a recombination intermediateJournal of Molecular Biology, 1999
- The χψ Subunits of DNA Polymerase III Holoenzyme Bind to Single-stranded DNA-binding Protein (SSB) and Facilitate Replication of an SSB-coated TemplateJournal of Biological Chemistry, 1998
- Holliday Junction Resolvases Encoded by HomologousrusAGenes inEscherichia coliK-12 and Phage 82Journal of Molecular Biology, 1996
- Hyper-recombination and Bloom's syndrome: microbes again provide clues about cancer.Genome Research, 1995
- Lethality of rep recB and rep recC double mutants of Escherichia coliMolecular Microbiology, 1995
- DNA helicases: Enzymes with essential roles in all aspects of DNA metabolismBioEssays, 1994
- The rep mutation: IV. Slower movement of replication forks in Escherichia coli rep strainsJournal of Molecular Biology, 1975