Erythrocyte Abnormality in Human Myopathy

Abstract
Erythrocyte ghosts isolated from myopathic patients responded to 10-4 molar ouabain with a dramatic increase in adenosine triphosphatase activity, while identical preparations from normal donors were inhibited by the same drug. These results have been interpreted in terms of a disease-related change in membrane integrity bearing upon function of the transport enzyme.

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