Leaky Scid Phenotype Associated with Defective V(D)J Coding End Processing in Artemis-Deficient Mice
- 1 December 2002
- journal article
- research article
- Published by Elsevier in Molecular Cell
- Vol. 10 (6), 1379-1390
- https://doi.org/10.1016/s1097-2765(02)00755-4
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Mutational Analysis of All Conserved Basic Amino Acids in RAG-1 Reveals Catalytic, Step Arrest, and Joining-Deficient Mutants in the V(D)J RecombinaseMolecular and Cellular Biology, 2002
- Does Artemis End the Hunt for the Hairpin-Opening Activity in V(D)J Recombination?Cell, 2002
- Hairpin Opening and Overhang Processing by an Artemis/DNA-Dependent Protein Kinase Complex in Nonhomologous End Joining and V(D)J RecombinationCell, 2002
- The Mre11 Complex Is Required for Repair of Hairpin-Capped Double-Strand Breaks and Prevention of Chromosome RearrangementsCell, 2002
- Separation-of-Function Mutants Reveal Critical Roles for RAG2 in Both the Cleavage and Joining Steps of V(D)J RecombinationMolecular Cell, 2001
- Joining-Deficient RAG1 Mutants Block V(D)J Recombination In Vivo and Hairpin Opening In VitroMolecular Cell, 2001
- DNA repair protein Ku80 suppresses chromosomal aberrations and malignant transformationNature, 2000
- Ku70 Is Required for DNA Repair but Not for T Cell Antigen Receptor Gene Recombination In VivoThe Journal of Experimental Medicine, 1997
- Identification of a nonsense mutation in the carboxyl-terminal region of DNA-dependent protein kinase catalytic subunit in the scid mouse.Proceedings of the National Academy of Sciences, 1996
- Coding joint formation of endogenous T cell receptor genes in lymphoid cells from scid mice: unusual P‐nucleotide additions in VJ‐coding jointsEuropean Journal of Immunology, 1991