Tumor necrosis factor-α and endothelial cells modulate Notch signaling in the bone marrow microenvironment during inflammation
- 1 May 2008
- journal article
- research article
- Published by Elsevier in Experimental Hematology
- Vol. 36 (5), 545-558.e1
- https://doi.org/10.1016/j.exphem.2007.12.012
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Stra13 regulates satellite cell activation by antagonizing Notch signalingThe Journal of cell biology, 2007
- Continuous Endothelial Cell Activation Increases Angiogenesis: Evidence for the Direct Role of Endothelium Linking Angiogenesis and InflammationJournal of Vascular Research, 2006
- Proangiogenic stimulation of bone marrow endothelium engages mTOR and is inhibited by simultaneous blockade of mTOR and NF-κBBlood, 2006
- Jagged1-dependent Notch signaling is dispensable for hematopoietic stem cell self-renewal and differentiationBlood, 2005
- Hematopoietic stem cell repopulating ability can be maintained in vitro by some primary endothelial cellsExperimental Hematology, 2004
- Induction of Notch signaling by tumor necrosis factor in rheumatoid synovial fibroblastsOncogene, 2003
- Identification of the haematopoietic stem cell niche and control of the niche sizeNature, 2003
- Osteoblastic cells regulate the haematopoietic stem cell nicheNature, 2003
- Notch Signaling: Cell Fate Control and Signal Integration in DevelopmentScience, 1999
- Interleukin 1 induces cultured human endothelial cell production of granulocyte-macrophage colony-stimulating factor.Journal of Clinical Investigation, 1987