Lesions to Schaffer collaterals prevent ischemic death of CA1 pyramidal cells
- 1 July 1986
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 68 (2), 169-174
- https://doi.org/10.1016/0304-3940(86)90136-9
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Calcium Overload in Selectively Vulnerable Neurons of the Hippocampus during and after Ischemia: An Electron Microscopy Study in the RatJournal of Cerebral Blood Flow & Metabolism, 1984
- Delayed Neuronal Recovery and Neuronal Death in Rat Hippocampus following Severe Cerebral Ischemia: Possible Relationship to Abnormalities in Neuronal ProcessesJournal of Cerebral Blood Flow & Metabolism, 1984
- Chronic failure of inhibition of the CA1 area of the hippocampus following kainic acid lesions of the CA3/4 areaBrain Research, 1984
- Slow transmission of neural activity in hippocampal area CA1 in absence of active chemical synapsesNature, 1984
- Regional cerebral blood flow and glucose metabolism following transient forebrain ischemiaAnnals of Neurology, 1982
- Temporal profile of neuronal damage in a model of transient forebrain ischemiaAnnals of Neurology, 1982
- A new model of bilateral hemispheric ischemia in the unanesthetized rat.Stroke, 1979
- Selective chromatolysis of neurons in the gerbil brain: A possible consequence of “epileptic” activity produced by common carotid artery occlusionAnnals of Neurology, 1979
- Aspartate and glutamate as possible transmitters of excitatory hippocampal afferentsNature, 1976
- Brain Lesions, Obesity, and Other Disturbances in Mice Treated with Monosodium GlutamateScience, 1969