Surprising Dependency for Retinoblastoma Protein in Ras-Mediated Tumorigenesis
Open Access
- 1 February 2006
- journal article
- review article
- Published by Taylor & Francis in Molecular and Cellular Biology
- Vol. 26 (4), 1-19
- https://doi.org/10.1128/mcb.26.4.1165-1169.2006
Abstract
No abstract availableThis publication has 54 references indexed in Scilit:
- The Retinoblastoma Protein Is Required for Ras-Induced Oncogenic TransformationMolecular and Cellular Biology, 2006
- Unique and overlapping functions of pRb and p107 in the control of proliferation and differentiation in epidermisDevelopment, 2004
- Acute mutation of retinoblastoma gene function is sufficient for cell cycle re-entryNature, 2003
- Dissecting the Contribution of p16INK4A and the Rb Family to the Ras Transformed PhenotypeMolecular and Cellular Biology, 2003
- RB and cyclin dependent kinase pathways: defining a distinction between RB and p16 loss in lung cancerOncogene, 2002
- cdk4 Deficiency Inhibits Skin Tumor Development but Does Not Affect Normal Keratinocyte ProliferationThe American Journal of Pathology, 2002
- Ras signalling is required for inactivation of the tumour suppressor pRb cell-cycle control proteinCurrent Biology, 1997
- Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4aCell, 1997
- Tumor suppressor genesCurrent Opinion in Genetics & Development, 1994
- Abundance and state of phosphorylation of the retinoblastoma susceptibility gene product in human colon cancerMolecular and Cellular Biochemistry, 1992