• 1 January 1976
    • journal article
    • research article
    • Vol. 30 (6), 929-933
Abstract
The immune mechanism involved in the recovery from and resistance to GPIC was studied. Guinea pigs were injected with a dose of cyclophosphamide (CY) (300 mg/kg wt) that inhibits antibody synthesis. Such treatment produced a cellular depletion in the B[bone marrow-derived]-cell area without producing an appreciable change in the T[thymus-derived]-cell area of the spleen and lymph nodes. CY treatment markedly delayed the appearance of secretory immunoglobulin A antibody to GPIC in the tears, and other classes of antibodies to GPIC and sheep erythrocyte in the serum. Recovery from infection was impaired, and a subsequent injection of CY prolonged the duration of infection. B cells may play an important role in the control of this infection.