Characterization of Cd-induced molecular events prior to cellular damage in primary rat hepatocytes in culture: Activation of the stress activated signal protein JNK and transcription factor AP-1
- 8 July 2004
- journal article
- Published by Wiley in Journal of Biochemical and Molecular Toxicology
- Vol. 18 (3), 133-142
- https://doi.org/10.1002/jbt.20018
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Mediation of cadmium‐induced oxidative damage and glucose‐6‐phosphate dehydrogenase expression through glutathione depletionJournal of Biochemical and Molecular Toxicology, 2003
- Novel mechanism of Vitamin E protection against cyclosporine A cytotoxicity in cultured rat hepatocytesBiochemical Pharmacology, 2002
- Molecular inhibitory mechanisms of antioxidant enzymes in rat liver and kidney by cadmiumToxicology, 2002
- MAPK-regulated transcription: a continuously variable gene switch?Nature Reviews Molecular Cell Biology, 2002
- Oxidative pathways in cardiovascular disease: roles, mechanisms, and therapeutic implicationsPharmacology & Therapeutics, 2001
- Reactive oxygen species in cell signalingAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 2000
- Stimulation of p38 Mitogen-activated Protein Kinase Is an Early Regulatory Event for the Cadmium-induced Apoptosis in Human Promonocytic CellsJournal of Biological Chemistry, 2000
- Regulation of antioxidant enzyme gene expression in response to oxidative stress and during differentiation of mouse skeletal muscleFree Radical Biology & Medicine, 1999
- Characterization of cadmium-induced apoptosis in rat lung epithelial cells: evidence for the participation of oxidant stressToxicology, 1999
- The Regulation of AP-1 Activity by Mitogen-activated Protein KinasesJournal of Biological Chemistry, 1995