Role of Eotaxin-1 (CCL11) and CC Chemokine Receptor 3 (CCR3) in Bleomycin-Induced Lung Injury and Fibrosis
- 31 December 2005
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 167 (6), 1485-1496
- https://doi.org/10.1016/s0002-9440(10)61235-7
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (HL28737, HL31963, HL52285)
This publication has 64 references indexed in Scilit:
- A Critical Role for Eosinophils in Allergic Airways RemodelingScience, 2004
- Chemokines in allergic airway diseaseCurrent Opinion in Pharmacology, 2003
- Intrinsic Defect in T Cell Production of Interleukin (IL)-13 in the Absence of Both IL-5 and Eotaxin Precludes the Development of Eosinophilia and Airways Hyperreactivity in Experimental AsthmaThe Journal of Experimental Medicine, 2002
- Resolution of Lung Inflammation by CD44Science, 2002
- Cell-Type-Dependent Induction of eotaxin and CCR3 by Ionizing RadiationBiochemical and Biophysical Research Communications, 2000
- Circulating, but not local lung, IL-5 is required for the development of antigen-induced airways eosinophilia.Journal of Clinical Investigation, 1998
- Targeted Disruption of the Chemokine Eotaxin Partially Reduces Antigen-induced Tissue EosinophiliaThe Journal of Experimental Medicine, 1997
- IL-5-Deficient Mice Have a Developmental Defect in CD5+ B-1 Cells and Lack Eosinophilia but Have Normal Antibody and Cytotoxic T Cell ResponsesImmunity, 1996
- RANTES and macrophage inflammatory protein 1 alpha induce the migration and activation of normal human eosinophil granulocytes.The Journal of Experimental Medicine, 1992
- Cytokine RANTES released by thrombin-stimulated platelets is a potent attractant for human eosinophils.The Journal of Experimental Medicine, 1992