Latent coeliac disease or coeliac disease beyond villous atrophy?
- 1 October 2007
- Vol. 56 (10), 1339-1340
- https://doi.org/10.1136/gut.2006.113084
Abstract
Coeliac disease is an autoimmune-mediated enteropathy triggered in genetically susceptible persons by the ingestion of a single dietary factor – wheat, rye and barley-derived gluten. The permanency of gluten intolerance was suggested in early studies, which showed that symptoms and intestinal lesions recurred usually within 2 years when gluten was reintroduced to the diet.1,2 Later it was recognised that the process of gluten-induced mucosal deterioration may take years or even decades in some individual cases.3,4Keywords
This publication has 27 references indexed in Scilit:
- Long-term follow-up of 61 coeliac patients diagnosed in childhood: evolution toward latency is possible on a normal dietGut, 2007
- In vivo targeting of intestinal and extraintestinal transglutaminase 2 by coeliac autoantibodiesGut, 2004
- Significance of intraepithelial lymphocytosis in small bowel biopsy samples with normal mucosal architectureAmerican Journal of Gastroenterology, 2003
- Dietary treatment of gluten ataxiaJournal of Neurology, Neurosurgery & Psychiatry, 2003
- Hla types in celiac disease patients not carrying the DQA1*05-DQB1*02 (DQ2) heterodimer: results from the european genetics cluster on celiac diseaseHuman Immunology, 2003
- Molecular Dissection of the Tissue Transglutaminase Autoantibody Response in Celiac DiseaseThe Journal of Immunology, 2001
- Celiac Disease Without Villous AtrophyDigestive Diseases and Sciences, 2001
- Concordance of Dermatitis Herpetiformis and Celiac Disease in Monozygous TwinsJournal of Investigative Dermatology, 2000
- Late Mucosal Relapse in a Boy with Coeliac Disease and Cow's Milk AllergyActa Paediatrica, 1986
- Lymphocytic Infiltration of Epithelium in Diagnosis of Gluten-sensitive EnteropathyBMJ, 1972