Redox-active iron mediates amyloid-β toxicity
Top Cited Papers
- 15 February 2001
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 30 (4), 447-450
- https://doi.org/10.1016/s0891-5849(00)00494-9
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Characterization of Copper Interactions with Alzheimer Amyloid β PeptidesJournal of Neurochemistry, 2000
- In Situ Oxidative Catalysis by Neurofibrillary Tangles and Senile Plaques in Alzheimer’s DiseaseJournal of Neurochemistry, 2000
- The Aβ Peptide of Alzheimer's Disease Directly Produces Hydrogen Peroxide through Metal Ion ReductionBiochemistry, 1999
- Amyloid‐β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative StressJournal of Neurochemistry, 1998
- Dramatic Aggregation of Alzheimer Aβ by Cu(II) Is Induced by Conditions Representing Physiological AcidosisJournal of Biological Chemistry, 1998
- Mechanisms of Neurotoxicity Associated with Amyloid β Deposition and the Role of Free Radicals in the Pathogenesis of Alzheimer's Disease: A Critical AppraisalChemical Research in Toxicology, 1997
- The Role of Iron in Beta Amyloid ToxicityBiochemical and Biophysical Research Communications, 1995
- Vitamin E protects nerve cells from amyloid βprotein toxicityBiochemical and Biophysical Research Communications, 1992
- Aggregation-related toxicity of synthetic β-amyloid protein in hippocampal culturesEuropean Journal of Pharmacology: Molecular Pharmacology, 1991
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990