c-Jun NH2-terminal Kinase Activation Leads to a FADD-dependent but Fas Ligand-independent Cell Death in Jurkat T Cells
Open Access
- 1 March 2001
- journal article
- Published by Elsevier
- Vol. 276 (11), 8350-8357
- https://doi.org/10.1074/jbc.m008431200
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Fas Preassociation Required for Apoptosis Signaling and Dominant Inhibition by Pathogenic MutationsScience, 2000
- Requirement of JNK for Stress- Induced Activation of the Cytochrome c-Mediated Death PathwayScience, 2000
- Diverse functions of JNK signaling and c-Jun in stress response and apoptosisOncogene, 1999
- Apoptosis by Death FactorCell, 1997
- Induction of Apoptosis by ASK1, a Mammalian MAPKKK That Activates SAPK/JNK and p38 Signaling PathwaysScience, 1997
- The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heatCurrent Biology, 1996
- Interaction between c-Rel and the Mitogen-activated Protein Kinase Kinase Kinase 1 Signaling Cascade in Mediating B Enhancer ActivationPublished by Elsevier ,1996
- Persistent Activation of c-Jun N-terminal Kinase 1 (JNK1) in γ Radiation-induced ApoptosisJournal of Biological Chemistry, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosisCell, 1995