Functions of the myosin ATP and actin binding sites are required for C. elegans thick filament assembly
- 1 January 1990
- Vol. 60 (1), 133-140
- https://doi.org/10.1016/0092-8674(90)90723-r
Abstract
No abstract availableThis publication has 68 references indexed in Scilit:
- MOLECULAR GENETICS OF NEMATODE MUSCLEAnnual Review of Genetics, 1989
- Genetic dissection of Drosophila myofibril formation: effects of actin and myosin heavy chain null alleles.Genes & Development, 1989
- Ifm(2)2 is a myosin heavy chain allele that disrupts myofibrillar assembly only in the indirect flight muscle of Drosophila melanogaster.The Journal of cell biology, 1988
- Myosin heavy-chain mutations that disrupt Caenorhabditis elegans thick filament assembly.Genes & Development, 1988
- Assembly Processes in Vertebrate Skeletal Thick Filament FormationAnnual Review of Biophysics, 1988
- Three-Dimensional Structure of an Oncogene Protein: Catalytic Domain of Human c-H- ras P21Science, 1988
- Regulation of non‐muscle myosin structure and functionBioEssays, 1987
- Structural significance of the GTP-binding domain of ras p21 studied by site-directed mutagenesis.Molecular and Cellular Biology, 1987
- Genetic studies of the lac repressorJournal of Molecular Biology, 1977
- THE GENETICS OF CAENORHABDITIS ELEGANSGenetics, 1974