Dopaminergic deficiency and delayed visual evoked potentials in humans
- 1 May 1982
- journal article
- research article
- Published by Wiley in Annals of Neurology
- Vol. 11 (5), 478-483
- https://doi.org/10.1002/ana.410110507
Abstract
Abnormal delay in visual evoked potential (VEP) could be normalized in 9 of 14 previously untreated parkinsonian patients under the influence of levodopa/carbidopa therapy. In 6 of 11 previously untreated schizophrenic patients the VEP latency became abnormal following therapy with agents known to cause dopaminergic blockade. Besides defects in retrobulbar conduction secondary to demyelination, synaptic dysfunction due to neurotransmitter deficiency or receptor blockade must also be considered as a mechanism underlying VEP delays in humans.Keywords
This publication has 18 references indexed in Scilit:
- Pattern‐reversal visual evoked potentials in the hereditary ataxias and spinal degenerationsAnnals of Neurology, 1981
- Dopamine depletion slows retinal transmissionExperimental Neurology, 1981
- THE INCIDENCE AND NATURE OF VISUAL PATHWAY INVOLVEMENT IN FRIEDREICH'S ATAXIABrain, 1980
- Correlations between transmitter metabolite concentrations in human ventricular cerebrospinal fluid and pattern visual-evoked potentialsBrain Research, 1980
- Visual evoked potentials in two forms of hereditary spastic paraplegiaElectroencephalography and Clinical Neurophysiology, 1980
- Visual evoked potentials and the visuogram in multiple sclerosisAnnals of Neurology, 1979
- Measurements of Visual Evoked Potentials in Parkinson's DiseaseBrain, 1978
- Uptake and efflux of 14 C‐dopamine in plateletsNeurology, 1975
- Visual Evoked Response in Diagnosis of Multiple SclerosisBMJ, 1973
- ParkinsonismNeurology, 1967