Molecular and cellular mechanisms of joint destruction in rheumatoid arthritis: two cellular mechanisms explain joint destruction?
- 1 March 1993
- journal article
- review article
- Published by BMJ in Annals Of The Rheumatic Diseases
- Vol. 52 (Supplement), S39-S47
- https://doi.org/10.1136/ard.52.suppl_1.s39
Abstract
No abstract availableThis publication has 106 references indexed in Scilit:
- Expression of perforin and granzymes in vivo: potential diagnostic markers for activated cytotoxic cellsImmunology Today, 1991
- Cathepsin B in synovial cells at the site of joint destruction in rheumatoid arthritisArthritis & Rheumatism, 1991
- Correlation of histopathological features of pannus with patterns of damage in different joints in rheumatoid arthritis.Annals Of The Rheumatic Diseases, 1991
- Occurrence of two germline-related rheumatoid factor idiotypes in rheumatoid arthritis and in non-rheumatoid seropositive individualsClinical and Experimental Immunology, 1990
- Vβ17 gene polymorphism in wild-derived mouse strains: Two amino acid substitutions in the Vβ17 region greatly alter T cell receptor specificityCell, 1990
- Susceptibility of the cartilage collagens types II, IX and XI to degradation by the cysteine proteinases, cathepsins B and LFEBS Letters, 1990
- Expression of a Zinc Finger Gene in HTLV-I- and HTLV-II-Transformed CellsScience, 1990
- Rheumatoid ArthritisNew England Journal of Medicine, 1990
- Infection of human T-lymphotropic virus type I to astrocytes in vitro with induction of the class II major histocompatibility complexNeuroscience Letters, 1988
- The shared epitope hypothesis. an approach to understanding the molecular genetics of susceptibility to rheumatoid arthritisArthritis & Rheumatism, 1987