Differential Accumulation of Proteoglycans and Hyaluronan in Culprit Lesions
Open Access
- 1 October 2002
- journal article
- research article
- Published by Wolters Kluwer Health in Arteriosclerosis, Thrombosis, and Vascular Biology
- Vol. 22 (10), 1642-1648
- https://doi.org/10.1161/01.atv.0000034021.92658.4c
Abstract
Objective— The importance of the extracellular matrix molecules versican, biglycan, decorin, and hyaluronan in plaque instability has not been recognized.Keywords
This publication has 24 references indexed in Scilit:
- Hyaluronan-binding Proteins: Tying Up the GiantJournal of Biological Chemistry, 2002
- Signaling Properties of Hyaluronan ReceptorsJournal of Biological Chemistry, 2002
- Versican V1 Proteolysis in Human Aorta in Vivo Occurs at the Glu441-Ala442 Bond, a Site That Is Cleaved by Recombinant ADAMTS-1 and ADAMTS-4Journal of Biological Chemistry, 2001
- Localization of Apoptotic Macrophages at the Site of Plaque Rupture in Sudden Coronary DeathThe American Journal of Pathology, 2000
- Differences in Hyaluronic Acid-mediated Functions and Signaling in Arterial, Microvessel, and Vein-derived Human Endothelial CellsJournal of Biological Chemistry, 2000
- Migration of bovine aortic smooth muscle cells after wounding injury. The role of hyaluronan and RHAMM.Journal of Clinical Investigation, 1995
- Localization and solubilization of hyaluronan and of the hyaluronan-binding protein hyaluronectin in human normal and arteriosclerotic arterial wallsAtherosclerosis, 1994
- Risk of thrombosis in human atherosclerotic plaques: role of extracellular lipid, macrophage, and smooth muscle cell content.Heart, 1993
- The role of proteoglycans in cell adhesion, migration and proliferationCurrent Opinion in Cell Biology, 1992
- PROTEOGLYCANS: STRUCTURES AND INTERACTIONSAnnual Review of Biochemistry, 1991