A systemic lupus erythematosus (SLE)‐like disease in mice induced by abnormal T‐B cell cooperation. Preferential formation of autoantibodies characteristic of SLE
- 1 January 1982
- journal article
- research article
- Published by Wiley in European Journal of Immunology
- Vol. 12 (2), 152-159
- https://doi.org/10.1002/eji.1830120210
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Capacity of genetically different T lymphocytes to induce lethal graft-versus-host disease correlates with their capacity to generate suppression but not with their capacity to generate anti-F1 killer cells. A non-H-2 locus determines the inability to induce lethal graft-versus-host disease.The Journal of Experimental Medicine, 1981
- Studies on the mechanism of drug sensitization: T-cell-dependent popliteal lymph node reaction to diphenylhydantoinClinical Immunology and Immunopathology, 1981
- Splenic immunoglobulin-secreting cells and their regulation in autoimmune mice.The Journal of Experimental Medicine, 1980
- Evidence for thyroglobulin-reactive T cells in good responder miceCellular Immunology, 1978
- Quantitation of precipitating antibodies to certain soluble nuclear antigens in sleArthritis & Rheumatism, 1977
- Immunoglobulin synthesis by peripheral blood cells in systemic lupus erythematosusArthritis & Rheumatism, 1975
- Allogeneic interactions in the immune response and tolerance: I. The allogeneic effect in unprimed rats and miceCellular Immunology, 1974
- Hypothesis: A model for generalised autoimmunityCellular Immunology, 1973
- Genetics of the immune response. I. The immune response to the phage fd in high and low responding inbred strains of miceEuropean Journal of Immunology, 1971
- Serological Overlap Between Lupus Erythematosus, Rheumatoid Arthritis, and Thyroid Auto-immune DiseaseBMJ, 1961