Mutation of a Conserved Amino Acid Residue (Tryptophan 1173) in the Tyrosine Kinase Domain of the IGF-I Receptor Abolishes Autophosphorylation but Does Not Eliminate Biologic Function
Open Access
- 1 February 1995
- journal article
- Published by Elsevier
- Vol. 270 (6), 2764-2769
- https://doi.org/10.1074/jbc.270.6.2764
Abstract
No abstract availableKeywords
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