Functional expression of “cardiac-type” Nav1.5 sodium channel in canine intracardiac ganglia
- 31 July 2006
- journal article
- research article
- Published by Elsevier in Heart Rhythm
- Vol. 3 (7), 842-850
- https://doi.org/10.1016/j.hrthm.2006.03.021
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Sodium current in human intestinal interstitial cells of CajalAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2003
- Localization of Nav1.5 sodium channel protein in the mouse brainNeuroReport, 2002
- Long QT syndrome, Brugada syndrome, and conduction system disease are linked to a single sodium channel mutationJournal of Clinical Investigation, 2002
- Nav1.5 underlies the ‘third TTX-R sodium current’ in rat small DRG neuronsMolecular Brain Research, 2002
- SCN5A is expressed in human jejunal circular smooth muscle cellsNeurogastroenterology & Motility, 2002
- An unexpected role for brain-type sodium channels in coupling of cell surface depolarization to contraction in the heartProceedings of the National Academy of Sciences, 2002
- Molecular biology of sodium channels and their role in cardiac arrhythmiasAmerican Journal Of Medicine, 2001
- Functional Nicotinic Acetylcholine Receptors That Mediate Ganglionic Transmission in Cardiac Parasympathetic NeuronsJournal of Neuroscience, 2000
- Effects of Channel Cytoplasmic Regions on the Activation Mechanisms of Cardiac versus Skeletal Muscle Na+ ChannelsBiophysical Journal, 1999
- Ionic Mechanisms Responsible for the Electrocardiographic Phenotype of the Brugada Syndrome Are Temperature DependentCirculation Research, 1999