Heat shock protein 60 autoimmunity and early lipid lesions in cholesterol-fed C57BL/6JBom mice during Chlamydia pneumoniae infection
- 31 December 2004
- journal article
- Published by Elsevier in Atherosclerosis
- Vol. 177 (2), 321-328
- https://doi.org/10.1016/j.atherosclerosis.2004.08.021
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Experimental Chlamydia pneumoniae infection in NIH/S mice: effect of reinoculation with chlamydial or cell preparation on culture, PCR and histological findings of lung tissueVaccine, 2002
- Evidence for infectious agents in cardiovascular disease and atherosclerosisThe Lancet Infectious Diseases, 2002
- Atherosclerosis as an autoimmune disease: an updateTrends in Immunology, 2001
- Hypercholesterolemia Exacerbates Virus-Induced Immunopathologic Liver Disease Via Suppression of Antiviral Cytotoxic T Cell ResponsesThe Journal of Immunology, 2001
- The atherogenic effects of chlamydia are dependent on serum cholesterol and specific to Chlamydia pneumoniaeJCI Insight, 1999
- Role of Heat Shock Proteins in Protection from and Pathogenesis of Infectious DiseasesClinical Microbiology Reviews, 1999
- Humoral Immune Response to Conserved Epitopes ofChlamydia trachomatisand Human 60‐kDa Heat‐Shock Protein in Women with Pelvic Inflammatory DiseaseThe Journal of Infectious Diseases, 1998
- Murine Models ofChlamydia pneumoniaeInfection and AtherosclerosisThe Journal of Infectious Diseases, 1997
- Chlamydial disease pathogenesis. The 57-kD chlamydial hypersensitivity antigen is a stress response protein.The Journal of Experimental Medicine, 1989
- Quantitative assessment of atherosclerotic lesions in miceAtherosclerosis, 1987