Immunoglobulin Isotype Switching Is Inhibited and Somatic Hypermutation Perturbed in UNG-Deficient Mice
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- 1 October 2002
- journal article
- research article
- Published by Elsevier in Current Biology
- Vol. 12 (20), 1748-1755
- https://doi.org/10.1016/s0960-9822(02)01215-0
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Altering the pathway of immunoglobulin hypermutation by inhibiting uracil-DNA glycosylaseNature, 2002
- AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversificationNature, 2002
- DNA Double-Strand BreaksThe Journal of Experimental Medicine, 2002
- Biphasic Kinetics of the Human DNA Repair Protein MED1 (MBD4), a Mismatch-specific DNA N-GlycosylaseJournal of Biological Chemistry, 2000
- Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)Cell, 2000
- Identification of a new uracil-DNA glycosylase family by expression cloning using synthetic inhibitorsCurrent Biology, 1999
- Hyperresponsive B Cells in CD22-Deficient MiceScience, 1996
- The SCID but Not the RAG-2 Gene Product Is Required for Sμ–Sε Heavy Chain Class SwitchingImmunity, 1996
- Cloning and Expression of Human G/T Mismatch-specific Thymine-DNA GlycosylaseJournal of Biological Chemistry, 1996
- The V(D)J recombination activating gene, RAG-1Cell, 1989