Scn3b knockout mice exhibit abnormal ventricular electrophysiological properties
Open Access
- 1 October 2008
- journal article
- review article
- Published by Elsevier in Progress in Biophysics and Molecular Biology
- Vol. 98 (2-3), 251-266
- https://doi.org/10.1016/j.pbiomolbio.2009.01.005
Abstract
No abstract availableKeywords
This publication has 71 references indexed in Scilit:
- Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humansJournal of Clinical Investigation, 2008
- Sodium channel Scn1b null mice exhibit prolonged QT and RR intervalsJournal of Molecular and Cellular Cardiology, 2007
- Pharmacological separation of early afterdepolarizations from arrhythmogenic substrate in ΔKPQ Scn5a murine hearts modelling human long QT 3 syndromeActa Physiologica, 2007
- SCN4B -Encoded Sodium Channel β4 Subunit in Congenital Long-QT SyndromeCirculation, 2007
- BACE1 regulates voltage-gated sodium channels and neuronal activityNature Cell Biology, 2007
- Effects of flecainide and quinidine on arrhythmogenic properties of Scn5a+/− murine hearts modelling the Brugada syndromeThe Journal of Physiology, 2007
- A quantitative analysis of the effect of cycle length on arrhythmogenicity in hypokalaemic Langendorff-perfused murine heartsPflügers Archiv - European Journal of Physiology, 2007
- Arrhythmogenic mechanisms in the isolated perfused hypokalaemic murine heartActa Physiologica, 2007
- Mechanisms of ventricular arrhythmogenesis in mice following targeted disruption of KCNE1 modelling long QT syndrome 5The Journal of Physiology, 2006
- Effects of flecainide and quinidine on arrhythmogenic properties of Scn5a+/Δ murine hearts modelling long QT syndrome 3The Journal of Physiology, 2006