Interactive regulation of signalling pathways in bone cells: Possible modulation of PGE2-stimulated adenylyl cyclase activity by protein kinase C
- 1 May 1990
- journal article
- research article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Vol. 1052 (2), 323-326
- https://doi.org/10.1016/0167-4889(90)90228-6
Abstract
No abstract availableThis publication has 23 references indexed in Scilit:
- Regulation of GH3 pituitary tumour-cell adenylate cyclase activity by activators of protein kinase CBiochemical Journal, 1989
- Phorbol ester induces loss of VIP stimulation of adenylate cyclase and VIP‐binding sites in HT29 cellsFEBS Letters, 1987
- Innervation of Periosteum and Bone by Sympathetic Vasoactive Intestinal Peptide-Containing Nerve FibersScience, 1986
- Phorbol esters enhance basal and stimulated adenylate cyclase activity in a pituitary cell lineBiochemical and Biophysical Research Communications, 1986
- Molecular mechanisms of receptor desensitization using the β-adrenergic receptor-coupled adenylate cyclase system as a modelNature, 1985
- Modulation of cyclic AMP accumulation in GH3 cells by a phorbol ester and thyroliberinBiochemical and Biophysical Research Communications, 1985
- The role of protein kinase C in cell surface signal transduction and tumour promotionNature, 1984
- Functional Receptors for Vasoactive Intestinal Peptide on Human Osteosarcoma Cells*Endocrinology, 1984
- Vasoactive Intestinal Peptide Stimulates Bone Resorption via a Cyclic Adenosine 3′,5′-Monophosphate-Dependent Mechanism*Endocrinology, 1983
- Collagenase production by synovial fibroblasts treated with phorbol myristate acetateArthritis & Rheumatism, 1979