Ubiquitination of RIP1 Regulates an NF-κB-Independent Cell-Death Switch in TNF Signaling
Open Access
- 1 March 2007
- journal article
- other
- Published by Elsevier in Current Biology
- Vol. 17 (5), 418-424
- https://doi.org/10.1016/j.cub.2007.01.027
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Activation of IKK by TNFα Requires Site-Specific Ubiquitination of RIP1 and Polyubiquitin Binding by NEMOMolecular Cell, 2006
- Sensing of Lys 63-linked polyubiquitination by NEMO is a key event in NF-κB activationNature Cell Biology, 2006
- NF-κB Is Essential for Induction of CYLD, the Negative Regulator of NF-κBJournal of Biological Chemistry, 2004
- The Kinase Activity of Rip1 Is Not Required for Tumor Necrosis Factor-α-induced IκB Kinase or p38 MAP Kinase Activation or for the Ubiquitination of Rip1 by Traf2Journal of Biological Chemistry, 2004
- Signalling pathways of the TNF superfamily: a double-edged swordNature Reviews Immunology, 2003
- Essential role for IKKγ/NEMO in TCR-induced IL-2 expression in Jurkat T cellsEuropean Journal of Immunology, 2003
- Activation of the IκB Kinase Complex by TRAF6 Requires a Dimeric Ubiquitin-Conjugating Enzyme Complex and a Unique Polyubiquitin ChainCell, 2000
- Activation of the NF-κB pathway by Caspase 8 and its homologsOncogene, 2000
- The Distinct Roles of TRAF2 and RIP in IKK Activation by TNF-R1Immunity, 2000
- Activation of a pro‐apoptotic amplification loop through inhibition of NF‐κB‐dependent survival signals by caspase‐mediated inactivation of RIPFEBS Letters, 2000