Ras Regulates NFAT3 Activity in Cardiac Myocytes
Open Access
- 1 February 2001
- journal article
- Published by Elsevier
- Vol. 276 (5), 3524-3530
- https://doi.org/10.1074/jbc.m004275200
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Signaling Pathways for Cardiac Hypertrophy and FailureNew England Journal of Medicine, 1999
- Regulation of Ras·GTP Loading and Ras-Raf Association in Neonatal Rat Ventricular Myocytes by G Protein-coupled Receptor Agonists and Phorbol EsterJournal of Biological Chemistry, 1999
- Stimulation of the p38 Mitogen-activated Protein Kinase Pathway in Neonatal Rat Ventricular Myocytes by the G Protein–coupled Receptor Agonists, Endothelin-1 and Phenylephrine: A Role in Cardiac Myocyte Hypertrophy?The Journal of cell biology, 1998
- Protection from reoxygenation injury by inhibition of rac1.Journal of Clinical Investigation, 1998
- Ras and Rho are Required for Gαq-Induced Hypertrophic Gene Expression in Neonatal Rat Cardiac MyocytesJournal of Molecular and Cellular Cardiology, 1998
- The Low Molecular Weight GTPase Rho Regulates Myofibril Formation and Organization in Neonatal Rat Ventricular MyocytesPublished by Elsevier ,1998
- The MEKK-JNK Pathway Is Stimulated by α1-Adrenergic Receptor and Ras Activation and Is Associated with in Vitro and in Vivo Cardiac HypertrophyJournal of Biological Chemistry, 1997
- TRANSCRIPTION FACTORS OF THE NFAT FAMILY:Regulation and FunctionAnnual Review of Immunology, 1997
- The small GTP-binding proteins Rac1 and Cdc42regulate the activity of the JNK/SAPK signaling pathwayCell, 1995
- Prognostic Implications of Echocardiographically Determined Left Ventricular Mass in the Framingham Heart StudyNew England Journal of Medicine, 1990