Inhibition by anaesthetics of 14C-guanidinium flux through the voltage-gated sodium channel and the cation channel of the 5-HT3 receptor of N1E-115 neuroblastoma cells
- 1 February 1993
- journal article
- research article
- Published by Springer Nature in Naunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie
- Vol. 347 (2), 125-132
- https://doi.org/10.1007/bf00169256
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Human brain sodium channels as one of the molecular target sites for the new intravenous anaesthetic propofol (2,6-diisopropylphenol)European Journal of Pharmacology: Molecular Pharmacology, 1991
- [14C]Guanidinium ion influx into Na+ channel preparations from mouse cerebral cortexEuropean Journal of Pharmacology: Molecular Pharmacology, 1990
- Molecular sites of anestheticaction in postsynaptic nicotinic membranesTrends in Pharmacological Sciences, 1989
- Sodium channel activation does not alter lipid metabolism in cultured neuroblastoma cellsNeurochemical Research, 1988
- Pharmacokinetics and Pharmacodynamics of Propofol Infusions during General AnesthesiaAnesthesiology, 1988
- Pharmacological characterization of serotonin 5-HT3 receptor-mediated electrical response in cultured mouse neuroblastoma cellsNeuropharmacology, 1988
- The Hemodynamic Consequences of High-dose Methohexital Anesthesia in HumansAnesthesiology, 1984
- Membrane interactions with general and local anaesthetics: A review of molecular hypotheses of anaesthesiaMolecular Aspects of Medicine, 1983
- DRUG INTERACTIONS AT THE GABA RECEPTOR-IONOPHORE COMPLEXAnnual Review of Pharmacology and Toxicology, 1982
- Pharmacokinetics of Etomidate, a New Intravenous AnestheticAnesthesiology, 1978