Inhibition of G-protein-coupled Inward Rectifying K+Channels by Intracellular Acidosis
Open Access
- 1 February 2003
- journal article
- Published by Elsevier
- Vol. 278 (9), 7091-7098
- https://doi.org/10.1074/jbc.m211461200
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Identification of Critical Residues Controlling G Protein-gated Inwardly Rectifying K+ Channel Activity through Interactions with the βγ Subunits of G ProteinsJournal of Biological Chemistry, 2002
- Eicosanoids Inhibit the G-protein-gated Inwardly Rectifying Potassium Channel (Kir3) at the Na+/PIP2 Gating SiteJournal of Biological Chemistry, 2001
- Activation of inwardly rectifying K+ channels by distinct PtdIns(4,5)P2 interactionsNature Cell Biology, 1999
- Identification of a Potassium Channel Site That Interacts with G Protein βγ Subunits to Mediate Agonist-induced SignalingPublished by Elsevier ,1999
- Direct activation of inward rectifier potassium channels by PIP2 and its stabilization by GβγNature, 1998
- Voltage‐gated and inwardly rectifying potassium channelsThe Journal of Physiology, 1997
- Evidence that direct binding of Gβγ to the GIRK1 G protein-gated inwardly rectifying K+ channel is important for channel activationNeuron, 1995
- Identification of domains conferring G protein regulation on inward rectifier potassium channelsCell, 1995
- Gβγ Directly Binds to the Carboxyl Terminus of the G Protein-Gated Muscarinic K+ Channel, GIRK1Biochemical and Biophysical Research Communications, 1995
- Arachidonic acid metabolites as intracellular modulators of the G protein-gated cardiac K+ channelNature, 1989