Neonatal Ventral Hippocampal Lesions Produce an Elevation of ΔFosB-Like Protein(s) in the Rodent Neocortex
Open Access
- 31 August 2005
- journal article
- research article
- Published by Springer Nature in Neuropsychopharmacology
- Vol. 31 (4), 700-711
- https://doi.org/10.1038/sj.npp.1300883
Abstract
No abstract availableKeywords
This publication has 55 references indexed in Scilit:
- Region-specific induction of ?FosB by repeated administration of typical versus atypical antipsychotic drugsSynapse, 1999
- Gene Expression for Glutamic Acid Decarboxylase Is Reduced Without Loss of Neurons in Prefrontal Cortex of SchizophrenicsArchives of General Psychiatry, 1995
- Subchronic Treatment with Haloperidol and Clozapine in Rats with Neonatal Excitotoxic Hippocampal DamageNeuropsychopharmacology, 1994
- Postpubertal Emergence of Hyperresponsiveness to Stress and to Amphetamine after Neonatal Excitotoxic Hippocampal Damage: A Potential Animal Model of SchizophreniaNeuropsychopharmacology, 1993
- Proliferative activation of quiescent Rat-1A cells by delta FosB.Molecular and Cellular Biology, 1993
- Distorted Distribution of Nicotinamide-Adenine Dinucleotide Phosphate—Diaphorase Neurons in Temporal Lobe of Schizophrenics Implies Anomalous Cortical DevelopmentArchives of General Psychiatry, 1993
- Altered Distribution of Nicotinamide-Adenine Dinucleotide Phosphate—Diaphorase Cells in Frontal Lobe of Schizophrenics Implies Disturbances of Cortical DevelopmentArchives of General Psychiatry, 1993
- Increased GABAA receptor binding in superficial layers of cingulate cortex in schizophrenicsJournal of Neuroscience, 1992
- Some Cytoarchitectural Abnormalities of the Entorhinal Cortex in SchizophreniaArchives of General Psychiatry, 1991
- Implications of Normal Brain Development for the Pathogenesis of SchizophreniaArchives of General Psychiatry, 1987