The higher toxicity of cereulide relative to valinomycin is due to its higher affinity for potassium at physiological plasma concentration
- 1 January 2006
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 210 (1-2), 39-46
- https://doi.org/10.1016/j.taap.2005.06.012
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Mitochondrial cytochrome c release may occur by volume-dependent mechanisms not involving permeability transitionBiochemical Journal, 2004
- In vitro assay for human toxicity of cereulide, the emetic mitochondrial toxin produced by food poisoning Bacillus cereusToxicology in Vitro, 2003
- Mitochondrial potassium transport: the K+ cycleBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2003
- Ionophoretic properties and mitochondrial effects of cereulideEuropean Journal of Biochemistry, 1999
- Fulminant Liver Failure in Association with the Emetic Toxin ofBacillus cereusNew England Journal of Medicine, 1997
- Valinomycin Induces Apoptosis of Ascites Hepatoma Cells (AH-130) in Relation to Mitochondrial Membrane Potential.Cell Structure and Function, 1997
- Synthesis and activity of cereulide, a cyclic dodecadepsipeptide ionophore as emetic toxin from Bacillus cereusBioorganic & Medicinal Chemistry Letters, 1995
- Membrane potential of mitochondria measured with an electrode sensitive to tetraphenyl phosphonium and relationship between proton electrochemical potential and phosphorylation potential in steady stateThe Journal of Membrane Biology, 1979
- Estimation of Membrane Potential and pH Difference across the Cristae Membrane of Rat Liver MitochondriaEuropean Journal of Biochemistry, 1969
- Mechanism of action of valinomycin on mitochondriaBiochemical and Biophysical Research Communications, 1964