DNA damage tumor suppressor genes and genomic instability
- 1 February 2004
- journal article
- review article
- Published by Elsevier in Current Opinion in Genetics & Development
- Vol. 14 (1), 11-16
- https://doi.org/10.1016/j.gde.2003.12.003
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 41 references indexed in Scilit:
- H2AX Haploinsufficiency Modifies Genomic Stability and Tumor SusceptibilityCell, 2003
- Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaksNature Cell Biology, 2003
- The mammalian mismatch repair protein MSH2 is required for correct MRE11 and RAD51 relocalization and for efficient cell cycle arrest induced by ionizing radiation in G2 phaseOncogene, 2003
- p53 Binding Protein 53BP1 Is Required for DNA Damage Responses and Tumor Suppression in MiceMolecular and Cellular Biology, 2003
- MDC1 is coupled to activated CHK2 in mammalian DNA damage response pathwaysNature, 2003
- The codon 72 polymorphic variants of p53 have markedly different apoptotic potentialNature Genetics, 2003
- Critical Role for Mouse Hus1 in an S-Phase DNA Damage Cell Cycle CheckpointMolecular and Cellular Biology, 2003
- 53BP1 functions in an ATM-dependent checkpoint pathway that is constitutively activated in human cancerNature Cell Biology, 2002
- 53BP1, a Mediator of the DNA Damage CheckpointScience, 2002
- The DNA damage-dependent intra–S phase checkpoint is regulated by parallel pathwaysNature Genetics, 2002