Peroxisome Proliferator-Activated Receptor (PPAR)γ Can Inhibit Chronic Renal Allograft Damage
- 31 May 2010
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 176 (5), 2150-2162
- https://doi.org/10.2353/ajpath.2010.090370
Abstract
No abstract availableKeywords
Funding Information
- Deutsche Forschungsgemeinschaft (SFB 405, B10; GR 880/3; SCHA 1082/2-1)
- European Union (INNOCHEM to H.-J.G.)
- National Research Program Tumor–Vessel Interface (SPP1190)
- National Aeronautics and Space Administration Specialized Center of Research (NNJ04HJ12G to A.A.)
This publication has 62 references indexed in Scilit:
- Biglycan, a Danger Signal That Activates the NLRP3 Inflammasome via Toll-like and P2X ReceptorsJournal of Biological Chemistry, 2009
- Deletion of PPARγ in Alveolar Macrophages Is Associated with a Th-1 Pulmonary Inflammatory ResponseThe Journal of Immunology, 2009
- Coordination of inflammation and metabolism by PPAR and LXR nuclear receptorsCurrent Opinion in Genetics & Development, 2008
- The renal cortical interstitium: morphological and functional aspectsHistochemistry and Cell Biology, 2008
- Molecular mechanisms of the antiangiogenic and antitumor effects of mycophenolic acidMolecular Cancer Therapeutics, 2008
- Cellular and molecular mechanisms of fibrosisThe Journal of Pathology, 2007
- PPAR‐γ in the Cardiovascular SystemPPAR Research, 2007
- The aetiology and pathogenesis of chronic allograft nephropathyTransplant Immunology, 2006
- Absence of Decorin Adversely Influences Tubulointerstitial Fibrosis of the Obstructed Kidney by Enhanced Apoptosis and Increased Inflammatory ReactionThe American Journal of Pathology, 2002
- PPARγ Ligand Inhibits Osteopontin Gene Expression Through Interference With Binding of Nuclear Factors to A/T-Rich Sequence in THP-1 CellsCirculation Research, 2002