SOCS1 Inhibits Tumor Necrosis Factor-induced Activation of ASK1-JNK Inflammatory Signaling by Mediating ASK1 Degradation
Open Access
- 1 March 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (9), 5559-5566
- https://doi.org/10.1074/jbc.m512338200
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Hsp90–Akt phosphorylates ASK1 and inhibits ASK1-mediated apoptosisOncogene, 2005
- SOCS-1 suppresses TNF- -induced apoptosis through the regulation of Jak activationInternational Immunology, 2004
- TheRole ofSuppressors ofCytokineSignaling(SOCS) Proteins inRegulation of theImmuneResponseAnnual Review of Immunology, 2004
- Type 1 Insulin-like Growth Factor Receptor (IGF-IR) Signaling Inhibits Apoptosis Signal-regulating Kinase 1 (ASK1)Journal of Biological Chemistry, 2003
- Etk/Bmx as a Tumor Necrosis Factor Receptor Type 2-Specific Kinase: Role in Endothelial Cell Migration and AngiogenesisMolecular and Cellular Biology, 2002
- SOCS Proteins: Negative Regulators of Cytokine SignalingThe International Journal of Cell Cloning, 2001
- Suppressors of Cytokine Signaling-1 and -6 Associate with and Inhibit the Insulin ReceptorJournal of Biological Chemistry, 2001
- Laminar flow inhibits TNF-induced ASK1 activation by preventing dissociation of ASK1 from its inhibitor 14-3-3JCI Insight, 2001
- Stat1 as a Component of Tumor Necrosis Factor Alpha Receptor 1-TRADD Signaling Complex To Inhibit NF-κB ActivationMolecular and Cellular Biology, 2000
- SOCS1 Is a Critical Inhibitor of Interferon γ Signaling and Prevents the Potentially Fatal Neonatal Actions of this CytokineCell, 1999