Neuropathic Sensitization of Behavioral Reflexes and Spinal NMDA Receptor/CaM Kinase II Interactions Are Disrupted in PSD-95 Mutant Mice
Open Access
- 1 February 2003
- journal article
- research article
- Published by Elsevier in Current Biology
- Vol. 13 (4), 321-328
- https://doi.org/10.1016/s0960-9822(03)00084-8
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Interaction with the NMDA receptor locks CaMKII in an active conformationNature, 2001
- Isolation of 2000-kDa complexes of N-methyl-d-aspartate receptor and postsynaptic density 95 from mouse brainJournal of Neurochemistry, 2001
- Mechanism and Regulation of Calcium/Calmodulin-dependent Protein Kinase II Targeting to the NR2B Subunit of the N-Methyl-d-aspartate ReceptorJournal of Biological Chemistry, 2000
- αCaMKII binding to the C‐terminal tail of NMDA receptor subunit NR2A and its modulation by autophosphorylationFEBS Letters, 1999
- Learning Mechanisms: The Case for CaM-KIIScience, 1997
- Characterization of Protein Kinase A and Protein Kinase C Phosphorylation of the N-Methyl-D-aspartate Receptor NR1 Subunit Using Phosphorylation Site-specific AntibodiesPublished by Elsevier ,1997
- Time course of degeneration and regeneration of myelinated nerve fibres following chronic loose ligatures of the rat sciatic nerve: can nerve lesions be linked to the abnormal pain-related behaviours?Pain, 1993
- The newly synthesized selective Ca2+calmodulin dependent protein kinase II inhibitor KN-93 reduces dopamine contents in PC12h cellsBiochemical and Biophysical Research Communications, 1991
- A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in manPain, 1988
- Lack of analgesic effect of opioids on neuropathic and idiopathic forms of painPain, 1988