ADAM17 deficiency by mature neutrophils has differential effects on L-selectin shedding
Open Access
- 1 October 2006
- journal article
- Published by American Society of Hematology in Blood
- Vol. 108 (7), 2275-2279
- https://doi.org/10.1182/blood-2006-02-005827
Abstract
L-selectin directs neutrophils to sites of inflammation, and upon their activation, surface expression of the receptor is rapidly down-regulated by eKeywords
This publication has 46 references indexed in Scilit:
- ADAM17 activity during human neutrophil activation and apoptosisEuropean Journal of Immunology, 2006
- Tumor Necrosis Factor-α-converting Enzyme (TACE/ADAM-17) Mediates the Ectodomain Cleavage of Intercellular Adhesion Molecule-1 (ICAM-1)Journal of Biological Chemistry, 2006
- L-Selectin Shedding Does Not Regulate Constitutive T Cell Trafficking but Controls the Migration Pathways of Antigen-activated T LymphocytesThe Journal of Experimental Medicine, 2003
- The monoclonal antibody CHO-131 binds to a core 2 O-glycan terminated with sialyl-Lewis x, which is a functional glycan ligand for P-selectinBlood, 2002
- Regulation of Membrane Metalloproteolytic Cleavage of L-selectin (CD62L) by the Epidermal Growth Factor DomainJournal of Biological Chemistry, 2001
- ADAM17 but Not ADAM10 Mediates Tumor Necrosis Factor-α and L-Selectin Shedding from Leukocyte MembranesAntisense and Nucleic Acid Drug Development, 2001
- An Essential Role for Ectodomain Shedding in Mammalian DevelopmentScience, 1998
- Purification of ADAM 10 from bovine spleen as a TNFα convertaseFEBS Letters, 1997
- Neutrophils roll on adherent neutrophils bound to cytokine-induced endothelial cells via L-selectin on the rolling cells.The Journal of Experimental Medicine, 1994
- Membrane proximal cleavage of L-selectin: identification of the cleavage site and a 6-kD transmembrane peptide fragment of L-selectin.The Journal of cell biology, 1994