Overexpression of Alzheimer's Disease Amyloid-β Opposes the Age-dependent Elevations of Brain Copper and Iron
Open Access
- 1 November 2002
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 277 (47), 44670-44676
- https://doi.org/10.1074/jbc.m204379200
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Treatment with a Copper-Zinc Chelator Markedly and Rapidly Inhibits β-Amyloid Accumulation in Alzheimer's Disease Transgenic MiceNeuron, 2001
- Oxidative stress in Alzheimer’s diseaseBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2000
- Aqueous Dissolution of Alzheimer's Disease Aβ Amyloid Deposits by Biometal DepletionJournal of Biological Chemistry, 1999
- Amyloid‐β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative StressJournal of Neurochemistry, 1998
- Increasing striatal iron content associated with normal agingMovement Disorders, 1998
- Zinc-induced Alzheimer's Aβ1–40 Aggregation Is Mediated by Conformational FactorsJournal of Biological Chemistry, 1997
- The Amyloid Precursor Protein of Alzheimer's Disease in the Reduction of Copper(II) to Copper(I)Science, 1996
- Rapid induction of Alzheimer A beta amyloid formation by zincScience, 1994
- The βA4 amyloid precursor protein binding to copperFEBS Letters, 1994
- Changes with age in copper and superoxide dismutase levels in brains of C57BL/6J miceMechanisms of Ageing and Development, 1979