Chapter 17 Chapter The nature and causes of hippocampal long-term potentiation
- 1 January 1990
- book chapter
- Published by Elsevier
- Vol. 83, 233-250
- https://doi.org/10.1016/s0079-6123(08)61253-4
Abstract
No abstract availableThis publication has 92 references indexed in Scilit:
- The NMDA receptor-mediated components of responses evoked by patterned stimulation are not increased by long-term potentiationBrain Research, 1989
- Postsynaptic Calcium Is Sufficient for Potentiation of Hippocampal Synaptic TransmissionScience, 1988
- Phorbol ester-induced synaptic potentiation differs from long-term potentiation in the guinea pig hippocampus in vitroNeuroscience Letters, 1988
- l‐Phenylalanyl‐l‐Glutamate‐Stimulated, Chloride‐Dependent Glutamate Binding Represents Glutamate Sequestration Mediated by an Exchange SystemJournal of Neurochemistry, 1987
- Subcellular compartmentalization of calcium-dependent and calcium-independent neutral proteases in brainSynapse, 1987
- Pharmacological dissociation of memory: Anisomycin, a protein synthesis inhibitor, and leupeptin, a protease inhibitor, block different learning tasksBehavioral and Neural Biology, 1985
- Regulation of glutamate receptor binding by the cytoskeletal protein fodrinNature, 1985
- The Ca2+/Cl− dependent L‐[3H]glutamate binding: a new receptor or a particular transport process?FEBS Letters, 1984
- First occurrence of hippocampal spatial firing in a new environmentExperimental Neurology, 1978
- Time-dependent changes in commissural field potentials in the dentate gyrus following lesions of the entorhinal cortex in adult ratsBrain Research, 1975