Jaundice in low birthweight infants: pathobiology and outcome
Open Access
- 1 November 2003
- journal article
- review article
- Published by BMJ in Archives of Disease in Childhood: Fetal & Neonatal
- Vol. 88 (6), 455F-458
- https://doi.org/10.1136/fn.88.6.f455
Abstract
Jaundice in preterm, as well as full term, infants results from (a) an increased bilirubin load in the hepatocyte, (b) decreased hepatic uptake of bilirubin from the plasma, and/or (c) defective bilirubin conjugation. Hyperbilirubinaemia in preterm infants is more prevalent, more severe, and its course more protracted than in term neonates.Keywords
This publication has 39 references indexed in Scilit:
- Treatment of jaundice in low birthweight infantsArchives of Disease in Childhood: Fetal & Neonatal, 2003
- Therapeutic Approaches to Neonatal Jaundice: An International SurveyClinical Pediatrics, 1996
- Clinical risk factors and periventricular leucomalacia.Archives of Disease in Childhood, 1988
- Toxicity to bilirubin in neonates: Infant development during first year in relation to maximum neonatal serum bilirubin concentrationThe Journal of Pediatrics, 1977
- Hyperbilirubinemia in premature infants: A follow-up studyThe Journal of Pediatrics, 1959
- Replacement Transfusion as a Means of Preventing Kernikterus of PrematurityArchives of Disease in Childhood, 1958
- A Study of Serum Bilirubin Levels in Relation to Kernikterus and PrematurityArchives of Disease in Childhood, 1956
- Kernicterus and PrematurityArchives of Disease in Childhood, 1955
- Increased Plasma Bilirubin in Newborn Infants in Relation to Birth WeightBMJ, 1954
- KERNICTERUS AND PREMATURITYThe Lancet, 1950