The Cell Death-promoting Gene DP5, Which Interacts with the BCL2 Family, Is Induced during Neuronal Apoptosis Following Exposure to Amyloid β Protein
Open Access
- 1 March 1999
- journal article
- Published by Elsevier
- Vol. 274 (12), 7975-7981
- https://doi.org/10.1074/jbc.274.12.7975
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Expression pattern of a novel death-promoting gene, DP5, in the developing murine nervous systemMolecular Brain Research, 1998
- Molecular Cloning of a Novel Polypeptide, DP5, Induced during Programmed Neuronal DeathPublished by Elsevier ,1997
- harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-XLThe EMBO Journal, 1997
- A Role for 4‐Hydroxynonenal, an Aldehydic Product of Lipid Peroxidation, in Disruption of Ion Homeostasis and Neuronal Death Induced by Amyloid β‐PeptideJournal of Neurochemistry, 1997
- BID: a novel BH3 domain-only death agonist.Genes & Development, 1996
- Induction of Apoptosis by Human Nbk/Bik, a BH3-Containing Protein That Interacts with E1B 19KMolecular and Cellular Biology, 1996
- β-Amyloid polypeptide increases calcium-uptake in PC12 cells: a possible mechanism for its cellular toxicity in Alzheimer's diseaseBrain Research, 1994
- Secreted Forms of β-Amyloid Precursor Protein Protect Hippocampal Neurons against Amyloid β-Peptide-Induced Oxidative InjuryExperimental Neurology, 1994
- Hydrogen peroxide mediates amyloid β protein toxicityCell, 1994
- beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicityJournal of Neuroscience, 1992