The Beta Cell in Autoimmune Diabetes: Many Mechanisms and Pathways of Loss
- 1 February 2000
- journal article
- review article
- Published by Elsevier in Trends in Endocrinology & Metabolism
- Vol. 11 (1), 11-15
- https://doi.org/10.1016/s1043-2760(99)00210-6
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Tumor Necrosis Factor- -Activated Cell Death Pathways in NIT-1 Insulinoma Cells and Primary Pancreatic CellsEndocrinology, 1999
- Cell Death in DevelopmentCell, 1999
- Local Expression of TNFα in Neonatal NOD Mice Promotes Diabetes by Enhancing Presentation of Islet AntigensImmunity, 1998
- IFN-gamma action on pancreatic beta cells causes class I MHC upregulation but not diabetes.Journal of Clinical Investigation, 1998
- Irreversible Inhibition of Metabolic Function and Islet Destruction after a 36-Hour Exposure to Interleukin-1Endocrinology, 1997
- Evidence for the presence of type I IL-1 receptors on β-cells of islets of LangerhansBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1997
- Interferon-γ Is Essential for Destruction of β Cells and Development of Insulin-dependent Diabetes MellitusThe Journal of Experimental Medicine, 1997
- Genetic absence of gamma-interferon delays but does not prevent diabetes in NOD miceDiabetes, 1996
- TNF-α and IFN-γ potentiate the deleterious effects of IL-1β on mouse pancreatic islets mainly via generation of nitric oxideCytokine, 1994
- Essential role for interferon-gamma and interleukin-6 in autoimmune insulin-dependent diabetes in NOD/Wehi mice.Journal of Clinical Investigation, 1991