Protein kinase B (PKB/Akt) is required for the completion of meiosis in mouse oocytes
- 1 February 2008
- journal article
- Published by Elsevier in Developmental Biology
- Vol. 314 (1), 215-223
- https://doi.org/10.1016/j.ydbio.2007.12.005
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- The PI3K‐Akt pathway promotes microtubule stabilization in migrating fibroblastsGenes to Cells, 2007
- Formin-2 is required for spindle migration and for the late steps of cytokinesis in mouse oocytesDevelopmental Biology, 2006
- The PI3K/Akt pathway is present and functional in the preimplantation mouse embryoDevelopmental Biology, 2005
- Essential role of protein kinase Bγ (PKBγ/Akt3) in postnatal brain development but not in glucose homeostasisDevelopment, 2005
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR ComplexScience, 2005
- Dwarfism, impaired skin development, skeletal muscle atrophy, delayed bone development, and impeded adipogenesis in mice lacking Akt1 and Akt2Genes & Development, 2003
- Multiple Phosphoinositide 3-Kinase-Dependent Steps in Activation of Protein Kinase BMolecular and Cellular Biology, 2002
- A Role for PI 3-Kinase and PKB Activity in the G2/M Phase of the Cell CycleCurrent Biology, 2002
- Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)Science, 2001
- Transforming activity and mitosis-related expression of the AKT2 oncogene: evidence suggesting a link between cell cycle regulation and oncogenesisOncogene, 1997