A Mitochondria-K+ Channel Axis Is Suppressed in Cancer and Its Normalization Promotes Apoptosis and Inhibits Cancer Growth
Top Cited Papers
Open Access
- 1 January 2007
- journal article
- research article
- Published by Elsevier in Cancer Cell
- Vol. 11 (1), 37-51
- https://doi.org/10.1016/j.ccr.2006.10.020
Abstract
No abstract availableKeywords
Funding Information
- Alberta Heritage Foundation for Medical Research
- Canadian Institutes of Health Research
- Canada Foundation for Innovation
This publication has 46 references indexed in Scilit:
- Energy Converting NADH: Quinone Oxidoreductase (Complex I)Annual Review of Biochemistry, 2006
- The Intrinsic Mitochondrial Membrane Potential of Colonic Carcinoma Cells Is Linked to the Probability of Tumor ProgressionCancer Research, 2005
- Superoxide targets calcineurin signaling in vascular endotheliumBiochemical and Biophysical Research Communications, 2005
- Gene therapy targeting survivin selectively induces pulmonary vascular apoptosis and reverses pulmonary arterial hypertensionJournal of Clinical Investigation, 2005
- NFAT proteins: key regulators of T-cell development and functionNature Reviews Immunology, 2005
- Mitochondria as cancer drug targetsTrends in Molecular Medicine, 2004
- NFATc3-Induced Reductions in Voltage-Gated K + Currents After Myocardial InfarctionCirculation Research, 2004
- Expression of voltage-gated potassium channels Kv1.3 and Kv1.5 in human gliomasNeuroscience Letters, 2003
- NFAT Transcription Factors Are Critical Survival Factors That Inhibit Cardiomyocyte Apoptosis During Phenylephrine Stimulation In VitroCirculation Research, 2003
- Membrane Depolarization Inhibits Kv1.5 Voltage-gated K+ Channel Gene Transcription and Protein Expression in Pituitary CellsJournal of Biological Chemistry, 1995