Vav Family GEFs Link Activated Ephs to Endocytosis and Axon Guidance
- 1 April 2005
- Vol. 46 (2), 205-217
- https://doi.org/10.1016/j.neuron.2005.03.019
Abstract
No abstract availableKeywords
This publication has 65 references indexed in Scilit:
- Eph-Dependent Tyrosine Phosphorylation of Ephexin1 Modulates Growth Cone CollapseNeuron, 2005
- Vav2 as a Rac-GDP/GTP Exchange Factor Responsible for the Nectin-induced, c-Src- and Cdc42-mediated Activation of RacPublished by Elsevier ,2005
- Rac-dependent trans-endocytosis of ephrinBs regulates Eph–ephrin contact repulsionNature Cell Biology, 2003
- EGF receptor mediates adhesion-dependent activation of the Rac GTPase: a role for phosphatidylinositol 3-kinase and Vav2Oncogene, 2003
- Mechanisms and functions of eph and ephrin signallingNature Reviews Molecular Cell Biology, 2002
- Vav proteins, adaptors and cell signalingOncogene, 2001
- Vav2 is required for cell spreadingThe Journal of cell biology, 2001
- THE EPHRINS AND EPH RECEPTORS IN NEURAL DEVELOPMENTAnnual Review of Neuroscience, 1998
- AL‐1‐induced Growth Cone Collapse of Rat Cortical Neurons is Correlated with REK7 Expression and Rearrangement of the Actin CytoskeletonEuropean Journal of Neuroscience, 1997
- Lerk2 (Ephrin-B1) Is a Collapsing Factor for a Subset of Cortical Growth Cones and Acts by a Mechanism Different from AL-1 (Ephrin-A5)Molecular and Cellular Neuroscience, 1997