Cytokine-induced nitric oxide inhibits mitochondrial energy production and induces myocardial dysfunction in endotoxin-treated rat hearts
- 1 September 2004
- journal article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 37 (3), 775-784
- https://doi.org/10.1016/j.yjmcc.2004.06.014
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Metabolic dysfunction and depletion of mitochondria in hearts of septic ratsJournal of Molecular and Cellular Cardiology, 2004
- Nitric oxide in septic shockBiochimica et Biophysica Acta (BBA) - Bioenergetics, 1999
- IMPAIRED ENERGY METABOLISM IN HEARTS OF SEPTIC BABOONSShock, 1999
- NITRIC OXIDE SYNTHASE IS NOT INVOLVED IN CARDIAC CONTRACTILE DYSFUNCTION IN A RAT MODEL OF ENDOTOXEMIA WITHOUT SHOCKShock, 1997
- NITRIC OXIDE SYNTHASE INHIBITION DOES NOT PREVENT CARDIAC DEPRESSION IN ENDOTOXIC SHOCKShock, 1995
- Abnormal contractile function due to induction of nitric oxide synthesis in rat cardiac myocytes follows exposure to activated macrophage-conditioned medium.Journal of Clinical Investigation, 1993
- Negative Inotropic Effects of Cytokines on the Heart Mediated by Nitric OxideScience, 1992
- Reevaluation of the role of cellular hypoxia and bioenergetic failure in sepsisPublished by American Medical Association (AMA) ,1992
- Septic shock: treatmentThe Lancet, 1991
- Functional compartmentation of ATP and creatine phosphate in heart muscleJournal of Molecular and Cellular Cardiology, 1970