A Role for Hypothalamic Malonyl-CoA in the Control of Food Intake
Open Access
- 1 December 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (48), 39681-39683
- https://doi.org/10.1074/jbc.c500398200
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- MONITORING ENERGY BALANCE: Metabolites of Fatty Acid Synthesis as Hypothalamic SensorsAnnual Review of Biochemistry, 2005
- C75, a Fatty Acid Synthase Inhibitor, Reduces Food Intake via Hypothalamic AMP-activated Protein KinaseJournal of Biological Chemistry, 2004
- AMP kinase and malonyl-CoA: targets for therapy of the metabolic syndromeNature Reviews Drug Discovery, 2004
- AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamusNature, 2004
- Long-term effects of a fatty acid synthase inhibitor on obese mice: food intake, hypothalamic neuropeptides, and UCP3Biochemical and Biophysical Research Communications, 2004
- C75, a Fatty Acid Synthase Inhibitor, Modulates AMP-activated Protein Kinase to Alter Neuronal Energy MetabolismJournal of Biological Chemistry, 2004
- Role of AMP-activated protein kinase in leptin-induced fatty acid oxidation in muscleBiochemical Society Transactions, 2003
- Neither Agouti-Related Protein nor Neuropeptide Y Is Critically Required for the Regulation of Energy Homeostasis in MiceMolecular and Cellular Biology, 2002
- Structure−Activity Studies of Cerulenin Analogues as Protein Palmitoylation InhibitorsJournal of Medicinal Chemistry, 1999
- Sensitivity to leptin and susceptibility to seizures of mice lacking neuropeptide YNature, 1996