Enhanced cytotoxicity of amyloid β-peptide by a complement dependent mechanism
- 4 July 1994
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 175 (1-2), 99-102
- https://doi.org/10.1016/0304-3940(94)91088-x
Abstract
No abstract availableKeywords
This publication has 8 references indexed in Scilit:
- Neurotoxicity of Human Amylin in Rat Primary Hippocampal Cultures: Similarity to Alzheimer's Disease Amyloid‐β NeurotoxicityJournal of Neurochemistry, 1993
- Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly stateJournal of Neuroscience, 1993
- Complement mRNA in the mammalian brain: Responses to Alzheimer's disease and experimental brain lesioningNeurobiology of Aging, 1992
- β-amyloid peptide in vitro toxicity: Lot-to-lot variabilityNeurobiology of Aging, 1992
- Complement gene expression in human brain: comparison between normal and Alzheimer disease casesMolecular Brain Research, 1992
- Anti‐inflammatory agents as a therapeutic approach to Alzheimer's diseaseNeurology, 1992
- In vitro aging of ß-amyloid protein causes peptide aggregation and neurotoxicityBrain Research, 1991
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990