“New” hepatic fat activates PPARα to maintain glucose, lipid, and cholesterol homeostasis
Top Cited Papers
- 1 May 2005
- journal article
- research article
- Published by Elsevier in Cell Metabolism
- Vol. 1 (5), 309-322
- https://doi.org/10.1016/j.cmet.2005.04.002
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- PGC-1 promotes insulin resistance in liver through PPAR-α-dependent induction of TRB-3Nature Medicine, 2004
- Disturbed Cholesterol Homeostasis in a Peroxisome-Deficient PEX2 Knockout Mouse ModelMolecular and Cellular Biology, 2004
- Liver fatty acid‐binding protein is required for high rates of hepatic fatty acid oxidation but not for the action of PPAR‐α in fasting miceThe FASEB Journal, 2003
- Dexamethasone induction of hypertension and diabetes is PPAR-α dependent in LDL receptor–null miceNature Medicine, 2003
- Evidence That Peroxisome Proliferator-activated Receptor α Is Complexed with the 90-kDa Heat Shock Protein and the Hepatitis Virus B X-associated Protein 2Journal of Biological Chemistry, 2003
- Peroxisome Proliferator-activated Receptor α (PPARα) Influences Substrate Utilization for Hepatic Glucose ProductionPublished by Elsevier ,2002
- Peroxisome proliferator–activated receptor α mediates the adaptive response to fastingJournal of Clinical Investigation, 1999
- Hormonal regulation of lipoprotein lipase activity from 5-day-old rat hepatocytesMolecular and Cellular Endocrinology, 1996
- Free fatty acid accumulation in secretagogue-stimulated pancreatic islets and effects of arachidonate on depolarization-induced insulin secretionBiochemistry, 1991
- Glucose-induced phospholipid hydrolysis in isolated pancreatic islets: quantitative effects on the phospholipid content of arachidonate and other fatty acidsBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1986