Restoration of podosomes and chemotaxis in Wiskott–Aldrich syndrome macrophages following induced expression of WASp
- 29 January 2002
- journal article
- Published by Elsevier in The International Journal of Biochemistry & Cell Biology
- Vol. 34 (7), 806-815
- https://doi.org/10.1016/s1357-2725(01)00162-5
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- The Verprolin-like Central (VC) Region of Wiskott-Aldrich Syndrome Protein Induces Arp2/3 Complex-dependent Actin NucleationJournal of Biological Chemistry, 2001
- Regulation of Actin Filament Network Formation Through ARP2/3 Complex: Activation by a Diverse Array of ProteinsAnnual Review of Biochemistry, 2001
- Actin machinery: pushing the envelopeCurrent Opinion in Cell Biology, 2000
- Signaling to Actin DynamicsThe Journal of cell biology, 1999
- THE WISKOTT-ALDRICH SYNDROME PROTEIN (WASP): Roles in Signaling and Cytoskeletal OrganizationAnnual Review of Immunology, 1999
- Intrinsic dendritic cell abnormalities in Wiskott-Aldrich syndromeEuropean Journal of Immunology, 1998
- A Role for Cdc42 in Macrophage ChemotaxisThe Journal of cell biology, 1998
- Wiskott–Aldrich Syndrome Protein, a Novel Effector for the GTPase CDC42Hs, Is Implicated in Actin PolymerizationCell, 1996
- Actin-Based Cell Motility and Cell LocomotionCell, 1996
- Rho, Rac, and Cdc42 GTPases regulate the assembly of multimolecular focal complexes associated with actin stress fibers, lamellipodia, and filopodiaCell, 1995