p38 MAPK Activation by NGF in Primary Sensory Neurons after Inflammation Increases TRPV1 Levels and Maintains Heat Hyperalgesia
Top Cited Papers
Open Access
- 1 September 2002
- Vol. 36 (1), 57-68
- https://doi.org/10.1016/s0896-6273(02)00908-x
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Neuronal Plasticity and Signal Transduction in Nociceptive Neurons: Implications for the Initiation and Maintenance of Pathological PainNeurobiology of Disease, 2001
- Neuronal Plasticity: Increasing the Gain in PainScience, 2000
- Impaired Nociception and Pain Sensation in Mice Lacking the Capsaicin ReceptorScience, 2000
- Neuronal Activity-Dependent Cell Survival Mediated by Transcription Factor MEF2Science, 1999
- eIF4 Initiation Factors: Effectors of mRNA Recruitment to Ribosomes and Regulators of TranslationAnnual Review of Biochemistry, 1999
- Making New ConnectionsNeuron, 1999
- The Cloned Capsaicin Receptor Integrates Multiple Pain-Producing StimuliNeuron, 1998
- The Phosphorylation of Eukaryotic Initiation Factor eIF4E in Response to Phorbol Esters, Cell Stresses, and Cytokines Is Mediated by Distinct MAP Kinase PathwaysJournal of Biological Chemistry, 1998
- The small GTP-binding proteins Rac1 and Cdc42regulate the activity of the JNK/SAPK signaling pathwayCell, 1995
- Nerve growth factor regulates expression of neuropeptide genes in adult sensory neuronsNature, 1989